What causes dandruff, and how do you get rid of it?

Malassezia is packed with everything you need.

Forget it, it's always in paradise.

In fact, Malassezia is a type of yeast that lives and eats on all of our scalp.

And in almost half of the population, their work causes dandruff.

So, why do some people have more dandruff than others?

And how can it be treated?

We may see ourselves as individuals, but we are truly colonial.

Our skin contains billions of bacteria.

Malassezia yeast builds up in our skin shortly after birth.

The follicles, the tiny holes that grow hair throughout our body, make living spaces popular. Malassezia loves these hiding places because they contain glands that produce sebum that is thought to soften and strengthen our hair.

Malassezia developed to absorb proteins and fats from the skin and because of the large number of sebum-producing follicles, our scalp is one of the most oily areas in our bodies and as a result, one of the most fertile. As the fungus absorbs oil on our scalp, dandruff may form.

This is because sebum is composed of both saturated and unsaturated acids. Satisfied fats are carefully packed together.

Empty fats, on the other hand, contain two bonds that create an unusual kink in their structure.

Fatty Acids

Malassezia consumes sebum by releasing an enzyme that releases all fatty acids.

But they only eat the full fat, leaving it behind. These abnormal residues soak into the skin and open up its barrier, allowing water to escape.

The body experiences this breakdown and reacts defensively, causing inflammation that causes dandruff.

It also increases the number of skin cells to repair the damaged barrier. Generally, the outside of our skin, or epidermis, is completely regenerated every two to three weeks,

Epidermal cells break down, protrude, die, and form a strong outer layer of skin, which gradually emerges from single cells too small to be seen.

But with dandruff, the cells come out quickly to repair the broken barrier, which means they do not ripen and do not break down properly.

Instead, they form large, oily lumps around the hair follicles that break down as visible flakes.

This is how Malassezia's appetite and our body's response to its products lead to dandruff.

Currently, the most effective way to get rid of dandruff is to apply antifungals to things like shampoos, which are applied directly to the scalp, to kill Malassezia.

For those suffering from dandruff, it usually comes and goes as the secretion of sebum varies throughout human life due to hormonal changes.

But despite the fact that Malassezia colonized everyone at the same rate, not everyone gets dandruff.

Some people are in great danger.

Why it is not clear.

Do people with dandruff have a specific genetic predisposition?

Is their skin barrier easily penetrated?

Scientists are currently investigating whether people with dandruff do, in fact,

they lose more water in their vessels, and whether this is what causes their skin cells to multiply.

Researchers are learning that Malassezia interacts with our immune system using tiny molecules, fatty acids called oxylipins that control inflammation.

If they can find a way to prevent inflammatory oxylipins and strengthen their anti-inflammatory properties, they can develop new therapies.

Scientists are also investigating whether our relationship with Malassezia is beneficial.

They think that dandruff, which may be uncomfortable and embarrassing for us, creates a reliable, fatty source of leaven.

But dandruff is not contagious or is very dangerous to our health.

And Malassezia seems to be at the forefront of protecting their environment, our skin, from some of the most dangerous viruses like Staphylococcus aureus.

Therefore, while scientists have come to the conclusion of many mysteries surrounding this phenomenon, it should be noted:

the scalp still scratches his head.